Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. However, our capacity to rescue SERT functionality in KI midbrain synaptoneurosomes by the inhibition of FAK signifies elevated FAK signaling downstream in the Pro32Pro33 mutant, as confirmed by greater pFAK localization in five-HT synapses. https://pro33login56665.win-blog.com/12803704/the-single-best-strategy-to-use-for-pro33